In summary, our data implicate a novel mechanism protecting against liver steatosis via an oxidative stress adaptive response that activates the AMPK pathway
As detailed in this review, MNPs can translocate across biological barriers and accumulate in cardiac tissues, where they initiate molecular cascades involving oxidative stress, mitochondrial dysfunction, inflammation, cellular senescence, and apoptosis
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The fact that most of the studies included only live-born infants and did not record or observe stillbirths, spontaneous abortions, or induced abortions due to severe malformations, potentially led to underestimations of the strength of the associations between SSRIs and fetal congenital malformations
